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Additionally, treatment of MDA-MB231 can-cer cells with 17AAG, which inhibits ATP binding to Hsp90, was found to releasemutant p53 from Hsp90, leading to rescue of its ubiquitination and degradation[ 23]

Additionally, treatment of MDA-MB231 can-cer cells with 17AAG, which inhibits ATP binding to Hsp90, was found to releasemutant p53 from Hsp90, leading to rescue of its ubiquitination and degradation[ 23].

The questionnaire had over 600 items completed by themother while the researcher was present. Certainly, the foremost concern for patients is their underlying diagnosis andits safe treatment.

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